While TNF-α, IFN-β, TSLP, SAA, and sVCAM-1 are known to promote growth or enable immune evasion in haematological tumours [5,38,42,43,44], the unique elevation of IP-10 in haematological malignancies is also noteworthy as it promotes angiogenesis and metastasis in solid tumours and has also been found in increased levels in chronic myelomonocytic leukaemia patients [45,46]. This evidence concerns the gene CXCL10 and monocytic leukemia.