The findings of the current study along with the literature support the notion that Calcium oscillations (perturbations in calcium signalling) result in apoptosis of cancer cells by reducing the influx of calcium [4] by ML218 HCl (T-type calcium channel blocker) and an overload of calcium [5] as a result of excessive influx of Calcium by the TRPV1 agonist capsaicin. Here, TRPV1 is linked to cancer.