On the molecular level, SFN induces oxidative stress in cells, increases the level of Fas ligand (FasL), activates caspase 8 and cleavage of BH3-interacting domain death agonist (Bid), and in consequence triggers apoptosis in androgen-independent prostate cancer cells (PC-3 and DU-145) [37]. This evidence concerns the gene FASLG and prostate cancer.