To determine the molecular background behind the decrease in pediatric cancer cells’ viability, proliferation and disturbances in cell cycle progression after treatment with ASBDs, we investigated alteration in intracellular signaling pathways essential for the survival and growth of tumor cells, including cascades of PI3K/Akt [55,56] and MAP (Mitogen-Activated Protein) kinases [57,58]. The gene discussed is AKT1; the disease is cancer.