These pathways require XPA–G, CSA-B, TTDA, and UV-stimulated scaffold protein A (UVSSA) [16], whose deficiencies are associated with several human autosomal recessive genetic diseases, such as xeroderma pigmentosum (XP), Cockayne syndrome (CS), trichothiodystrophy (TTD), and UV sensitive syndrome (UVSS), respectively [16]. This evidence concerns the gene UVSSA and Cockayne syndrome.