Densitometric analyses of the protein blots revealed that, both in the LFPI and the rmTBI experiments, there were no changes in the hippocampal levels of the investigated microglial markers, specifically Iba1, iNOS, TLR-2, CD86, and CD206, at 14 d after both brain trauma protocols in relation to their designated control groups (Figure S5). Here, CD86 is linked to brain injury.