VEGF-C, which promotes lymphangiogenesis in the skin, was decreased in bronchoalveolar lavage fluid (BALF) of IPF patients compared to healthy volunteers, and lung fibrosis in the bleomycin-induced mouse model was reduced when lymphangiogenesis was induced by overexpression of VEGF-C [89]. The gene discussed is VEGFC; the disease is pulmonary fibrosis.