Previously, IL17B was shown to be overexpressed in breast cancer tissue and inversely correlate with breast cancer patient survival rate [122], and a recent study indicates that IL17B induces autophagosome formation in gastric cancer CSCs; in contrast, autophagy inhibition through ATG7 deletion inhibits IL17B-induced self-renewal, which draws a connection between autophagy and CSC maintenance [123]. The gene discussed is IL17B; the disease is breast cancer.