In this frame, the case described by Bor and colleagues [81] is intriguing: they exome sequenced a Danish family with thrombotic episodes, revealing from one side the dysfibrinogenemia-causing mutation Aα-p.Arg19Gly and from the other a series of single-nucleotide polymorphisms located in FGA, FGB, and FGG. Here, FGB is linked to familial hypodysfibrinogenemia.