Although all patients with COVID-19 had increased lactate dehydrogenase, eosinophil sedimentation rate, CRP, serum ferritin, IL-6, and D-dimer, which are all associated with infection and cardiac damage, patients with hypertension had higher neutrophils and neutrophil–lymphocyte ratios, ALT, creatinine, and fibrinogen compared with non-hypertensive patients, indicating that COVID-19 patients with hypertension tend to show more severe inflammation and organ damage than in patients without hypertension (Table 3) [54]. The gene discussed is IL6; the disease is COVID-19.