SETD1A and infection: In B. bassiana, abolished H3K4me1/ me2/me3 and attenuated H3K36me2 concurred in the absence of set1, leading to the nearly or completely abolished expression of cre1, hyd1 and hyd2, sharp repression of brlA and abaA as key activator genes of central development pathway (CPD), and severe defects in conidiation, host infection, proliferation in vivo and virulence [39].