The biological processes included regulation of sterol regulatory-element binding protein (SREBP) signaling, phospholipid transport, as well as granulocyte differentiation, which are implicated in controlling lipogenesis and lipid uptake, and are known to contribute to the development of NAFLD and nonalcoholic steatohepatitis [95]. This evidence concerns the gene CNBP and metabolic dysfunction-associated steatotic liver disease.