In the light of significance of this heat shock protein for cancer adaptability to endogenous (e.g., oxidative and metabolic stress, hypoxia) and exogeneous (e.g., chemo/radiotherapy) stressors as well as its role in stabilization of oncogenic proteins, particularly those facilitating cancer growth, invasion and metastasis [23,51], markedly weaker effect of oxicam analogues on HSP90AA1 protein is disappointing. The gene discussed is HSP90AA1; the disease is cancer.