As seen in our general low‐risk study population, elevated levels of a marker of left ventricular dysfunction and haemodynamic stress (NT‐proBNP) at baseline were associated with the development of AF in subjects with CKD, whereas markers of inflammation (hs‐CRP) and myocardial damage (hs‐cTnI) were not. The gene discussed is CRP; the disease is chronic kidney disease.