Compared with normal tissues, RPL23A, EFNB1, NES, and EPHB4 expression markedly increased in GBM tissues, while CALB2, ENO2, OBSCN, RAB9B, and STMN2 were downregulated in GBM cases (Figures 4(a)–4(j)). Here, EFNB1 is linked to glioblastoma.