RPS8 and infection: Collectively, these loci explain the majority of the phenotypic variation for colonization by Pst and loss of all three loci leads to infection (pustule formation) by Pst. Rps6, Rps7, and Rps8 collectively explained 62 and 67% of phenotypic variation with respect to chlorosis (CHL) and colonization resistance (pCOL), but only 32 and 30% of infection (INF) and pustule formation (pPUST) (Supplementary Data 2).