The imbalance of Th1/Th2 cytokines has been found as a predominant factor associated with asthma, where increased expression of Th2 cytokines, mainly IL-4, IL-5, IL-9, and IL-13 promote the serum immunoglobulin E (IgE) and eosinophilia that stimulate a variety of cellular processes, including mucus hypersecretion, airway inflammation, and hypersensitivity (Ngoc et al., 2005; Zhu et al., 2016). The gene discussed is IL5; the disease is asthma.