FOXO3 and pulmonary fibrosis: The reduced expression of miR-29c is observed in alveolar epithelial cells from IPF patients, and overexpression of miR-29c in mice model reduces apoptosis, increases epithelial renewal, and thereby reduces lung fibrosis through targeting forkhead box O3a (Foxo3a), which is a transcription factor and play a crucial role in the induction of apoptosis (Xie et al., 2017).