Since excessive alcohol exposure can cause cerebellar ataxia and locomotion dysregulation, such as impaired postural stability and balance as well as slower attenuated foot tapping, we assessed the levels of Zfp212, GAD65/67, and Pld3 in the Cb of mice administered 500 μL of alcohol (1–33.5%) per day for 1 week (Fig. 6a). This evidence concerns the gene PLD3 and cerebellar ataxia.