Notably, HOXATs, such as HOTAIRM1, HOXA11-AS, HOXB-AS1, HOXB-AS4, HAGLR, and HOXC-AS1, showed significant correlation with immune cell infiltration and were likely to exhibit higher expression levels in immune cells, while the correlation between their expression levels and tumor dryness scores suggested that they possibly act primarily as tumor promoters in the TME. This evidence concerns the gene HAGLR and neoplasm.