Our GSEA results demonstrated that ECM receptor interaction, focal adhesion, MAPK signaling pathway, pathways in cancer, and TGF-β signaling pathway were positively related to the high-risk scores while base excision repair, DNA replication, nucleotide excision repair, and pyrimidine metabolism were enriched in low-risk samples, indicating that varying prognosis among patients might be related to pathways. The gene discussed is TGFB1; the disease is cancer.