LINC00665 and hepatocellular carcinoma: In HCC, Shan and Li [19] reported that LINC00665 was overexpressed in HCC cells and tissue samples, which associated with poor prognosis of HCC patients; and LINC00665 depletion inhibited viability and induced apoptosis and autophagy through targeting miR-186-5p/MAP4K3 axis.