In line with previous reports in similar models but not expressing CXCL11 (Lefort et al., 2001; Lax et al., 2014), inhalation of nebulized LPS in DBA/1 mice induced key parameters recommended by the American Thoracic Society to detect the presence of ARDS in laboratory animals: LPS inhalation caused a rapid and significant recruitment of inflammatory cells to the alveolar space, especially neutrophils at early time points after LPS challenge followed by macrophages and T cells at later time points. This evidence concerns the gene CXCL11 and acute respiratory distress syndrome.