Palmitoylation of Wnt/β-catenin pathway is identified as a potential mediator of FASN oncogenicity in the prostate cancer, that is, overexpression of FASN increases incorporation of de novo palmitate into Wnt-1, and enhanced Wnt-1 palmtoylation in prostate epithelial cells causes cytoplasmic β-catenin accumulation and pathway activation 94. Here, FASN is linked to prostate carcinoma.