Thus, AUDA could block the inflammatory response in Kawasaki disease.[70] Treatment with AUDA reduced the protein expression levels of TNF‐α, matrix metalloproteinase, and IL‐1 in the Kawasaki disease mouse model, and the vascular repair by human coronary arterial endothelial cells (HCAECs) was markedly increased. The gene discussed is TNF; the disease is Kawasaki disease.