A recent study in mdx mice showed that CX43 is involved in the pathogenesis of DMD cardiomyopathy and that hypophosphorylation at specific serine residues (S325/S328/S330) is related to changes in CX43 expression, distribution and function in DMD cardiomyopathy (Himelman et al., 2020). The gene discussed is GJA1; the disease is cardiomyopathy.