It means that SA can be widely involved in immune regulation of ovarian cancer through SUCNR1, with mechanisms associated with multiple biological processes such as T cell activation, interleukin signaling, chemokine signaling, antigen processing and delivery, natural killer cell-mediated cytotoxicity, PD-1-blocked cancer immunotherapy, adaptive immune system and interferon α/β signaling, etc. The prognosis of ovarian cancer patients with high expression of SUCNR and large infiltration of neutrophils is poor. This evidence concerns the gene SUCNR1 and ovarian cancer.