Various factors such as heredity including APOL1 gene variations [5], infection [6], hypertension [7], inflammatory diseases such as systemic lupus erythematosus (SLE) [8] and inflammatory bowel disease [9], drug-related side effects, and unknown causes [10] have been involved in the pathogenesis of the disease. This evidence concerns the gene APOL1 and systemic lupus erythematosus.