More studies have been conducted to improve experimental uveitis by inhibiting the NF-κB signaling pathway, such as with lutein (Izumi-Nagai et al., 2007; Kijlstra et al., 2012), growth hormone (Liang et al., 2020), aminooxy-acetic acid (Meka et al., 2015; Mei et al., 2020), dehydroxymethylepoxyquinomicin (Ando et al., 2020), astaxanthin (Suzuki et al., 2006), silibinin (Chen et al., 2017), and aryl hydrocarbon receptor (Huang et al., 2018). The gene discussed is NFKB1; the disease is uveitis.