Although limited by sparse lymphocytic renal infiltration in healthy controls, differential expression analysis within individual clusters revealed decreased expression of AIM1 and IL7R and increased expression of KLF2 in kidney-resident TFH cells from SLE patients compared to healthy controls, as well as increased expression of CX3CR1 in kidney-resident CD8 cells (Supplementary Fig. 2). The gene discussed is KLF2; the disease is systemic lupus erythematosus.