The results showed that there was no significant difference in cell proliferation in cells overexpressed LncFEZF1-AS1 plus si-ATG5 compared to cells with si-ATG5, indicating that cell proliferation promoted by FEZF1-AS1 was inhibited after knockdown of ATG5 in SGC-7901 GC cells (Figure 5A). The gene discussed is FEZF1; the disease is gastric cancer.