DDIT3 and acute myeloid leukemia: Previous studies demonstrated that baicalin attenuated ER stress induced by tunicamycin through the CHOP/eNOS/NO pathway in cardiomyocytes and prevented lipotoxicity in AML-12 cells by reducing the interactions between TXNIP and the NLRP3 inflammasome [30–33]; here, we also showed this effect.