In comparison with the ASFV-WT-infected PAMs, the majority of the dysregulated mRNAs during ASFV-ΔH240R infection were involved in innate immunity signaling (including TNF, MAPK, and JAK-STAT signaling pathways), receptor signaling, metabolic networks, and other pathways involved in viral infection. The gene discussed is SOAT1; the disease is viral infectious disease.