Given the ubiquity of SARS-CoV-2 receptors and its ability to infect a broad range of cell types [13], analysis of PTAFR activation patterns in infected tissues (including vascular endothelial cells) could provide further insights into increased platelet aggregation (via PAF signaling) and the risk for arterial thrombosis seen in severe/acute COVID-19 patients [16]. This evidence concerns the gene PTAFR and Venous thrombosis.