Consistent with a chronic inflammatory state, the serum levels of pro-inflammatory cytokines such as TNFα (Fig 2A), IFNγ (Fig 2B), and IL-1β (Fig 2C) increased as a result of the infection in the WT and FPR2-/- mice, although the IL-1β increase in the FPR2-/- mice was not significant. The gene discussed is IFNG; the disease is infection.