S100A8 and vulvovaginal candidiasis: In a previous study, S100A8 was reported to be the major antifungal component in neutrophil extracellular traps (NETs); its absence in NETs resulted in the complete loss of antifungal activity [35]; however, further study is necessary to clarify an exact protective role of α-GalCer-stimulated iNKT cells against our vulvovaginal candidiasis model.