In contrast, glycoproteins involved in proteasome function (e.g. PSMD4)49,50, N-linked glycosylation (e.g., ENTPD5)51–54, glycan metabolism (e.g., NAGA)55, and receptor trafficking (e.g., LAMP1)56 were upregulated primarily in localized PCa and mPCa (Fig. 3C, upper image). The gene discussed is NAGA; the disease is posterior cortical atrophy.