Dysfunction of vascular SMCs and other pathological cellular processes, such as inflammatory and immune responses, and vascular extracellular matrix remodeling, can contribute to AAA formation, and has been shown to be associated with LncRNAs, such growth-arrest-specific transcript 5 (GAS5), H19, Lnc-HLTF-5, HIF1α-as1 [8, 11]. This evidence concerns the gene GAS5 and triple-A syndrome.