On the molecular level, the outcome of the reciprocal translocation t (9;22) (q34q11) in CML is the formation of an oncogene termed BCR/ABL, which encodes a chimeric protein, BCR-ABL, with constitutive tyrosine kinase activity (Sun et al. 2013). Here, ABL1 is linked to chronic myelogenous leukemia, BCR-ABL1 positive.