In contrast to this, pathways which promote tumor growth and drug resistance (such as Wnt target, obesity acid elongation, drug metabolism by cytochrome P450 and fatty acid biosynthesis), showed significantly more activity in the CTNNB1-MUT group than in the CTNNB1-WT group (all P < 0.05; Figure 6B; Table S4). Here, CTNNB1 is linked to neoplasm.