However, we could not study the possible involvement of hippocampal CaMKII on SDIA-memory destabilization because pre-recall intra-CA1 AIP administration hinders memory expression (Additional file 3: Fig. S2) and post-reactivation intra-CA1 AIP is amnestic, which prevented us to analyze whether hippocampal CaMKII inhibition impedes the amnesia provoked by protein synthesis inhibitors. Here, CAMK2G is linked to amnesia.