TCL1A and non-Hodgkin lymphoma: Across non-Hodgkin lymphomas, a gene set enrichment analysis associated high TCL1A levels with important pathways controlling B-cell lymphomagenesis, including, e.g., B-cell receptor, NF-κB signaling, cell death, and MAP kinase, implicating a central role of elevated TCL1A expression in their pathogenesis and aggressiveness [38].