These mechanisms include the regulation of autocrine and paracrine loops critical for remodeling of the tumor microenvironment and tumor growth [25], nucleolar functions connected with either efficient ribogenesis or the suppression of nucleolar stress in cancer cells [26,27,28], the regulation of both the centrosome and chromosome stability [27,29], the regulation of the YAP/TAZ pathway [30], and the recruitment of pathways to avoid antitumoral immune responses [31]. Here, YAP1 is linked to neoplasm.