Additionally, the glycosylation level of apolipoprotein A-I allowed for the separation of both early and late stage AC from all other subgroups (except late stage AC compared with late stage SCLC) and statistically significant differences in glycosylation of albumin were also observed for late stage SCLC and all other LC subtypes except early stage SCC (Table 3 and Table S5). This evidence concerns the gene APOA1 and laryngotracheoesophageal cleft.