The restraint of myeloperoxidase by EGCG at ten mg/kg for five days uncovered the antagonist of RA benefits in pristane-induced arthritis (PIA), myeloperoxidase (MPO) [52], CTR, carbonic anhydrase II, cathepsins K, alpha- and beta-integrins, and NF-ATc1 all negatively responded in human osteoclasts and mice after 15 days of treatment at 20 and 50 M [53]. The gene discussed is MPO; the disease is arthritic joint disease.