IL6 and COVID-19: In their work, Magro et al. [32] also found increased levels of several cytokines related to severe COVID-19 forms (IL-6, TNF-α, IL-1β, IL-8, p38, and INF-γ) in the lungs within areas of viral proliferation; an increase in caspase-3 and programmed death-ligand 1 (PDL1) was also observed in the pulmonary endothelia containing infectious virus.