In [31], the authors discovered higher numbers of ACE-2-positive endothelial cells as well as significant changes in endothelial morphology, including cell swelling, disruption of intercellular junctions, and cell death, in post-mortem lung tissues from patients who died from COVID-19 or acute respiratory distress syndrome caused by influenza A (H1N1) infection, compared to age-matched, uninfected control lungs. Here, ACE2 is linked to acute respiratory distress syndrome.