MUL1 and stroke disorder: More evidence for miR-652 regulation of mitochondrial hemodynamics following ischemia was demonstrated in a study in which elevated NR4A2 expression (a transcription factor that regulates mitochondrial homeostasis and neuronal survival) after transient global cerebral ischemia decreased miR-652 expression but increased the mitochondrial E3 ubiquitin ligase 1 (Mul1) protein (known for regulating mitochondrial dynamics following stroke), thereby repressing cellular viability [78].