Hafeez et al. [34] observed that delphinidin suppressed cell growth arrest and triggered caspase-dependent death in prostate cancer cells (PC-3) in vitro in a dose-dependent manner by lowering phosphorylations of IκB kinase γ, NF-κB inhibitory protein IκB-α, and restricting NF-κB DNA binding activity. This evidence concerns the gene NFKB1 and prostate cancer.