NR3C1 and COVID-19: The dysregulated immune response observed in COVID-19 is qualitatively similar to that of multifactorial ARDS [4], in whom administration of methylprednisolone was shown to rescue the cellular concentrations and functions of the activated glucocorticoid receptor, leading to downregulation of systemic and pulmonary markers of inflammation, coagulation, and fibroproliferation [15,30].