In the repeated muscle biopsies of patients with dermatomyositis and on the lymphocytes or sera of patients with GBS who improved after IVIg [52, 53, 57, 59] there was impressive downregulation in a number of the aforementioned molecules including MHC-I, ICAM-I, LFA-1, TNF-α, IL1β, TGF-β, MHC-I, IP-10, Mig, MMP-2, and MMP-9 (Fig. 4A–D). The gene discussed is CXCL9; the disease is dermatomyositis.